Deletion of 22q11, a schizophrenia-associated chromosomal region, may activate previously silent mutations, according to unpublished work presented Sunday at the 2011 Society for Neuroscience annual meeting in Washington, D.C.

Researchers have developed a computational technique that allows them to simultaneously use multiple types of information — including gene expression, ratings and associations — to identify candidate genes for a disorder. The unpublished results were presented Sunday at the 2011 Society for Neuroscience annual meeting in Washington, D.C.

Mice lacking the autism risk gene SHANK2 show social deficits and are extremely hyperactive, according to unpublished research presented Saturday at the 2011 Society for Neuroscience annual meeting in Washington, D.C.

In a video interview at the 2011 Society for Neuroscience annual meeting in Washington, D.C., Janine LaSalle makes a case for the importance of the methylome in autism research.

Deleting the Rett syndrome gene in a subset of neurons, instead of throughout the body, dramatically lowers the number of genes that are dysregulated in those neurons, according to results presented in a poster session Saturday at the 2011 Society for Neuroscience annual meeting in Washington, D.C.

Male zebra finches have high levels of contactin associated protein-like 2 (CNTNAP2), a protein that has been linked to autism and language disorders, in a key song-related area of their brains, according to unpublished research presented in a poster session Sunday at the 2011 Society for Neuroscience annual meeting in Washington, D.C.

Wenbao Gan describes the technique he has devised to track the development of neurons in live mouse brains.

A subset of people with autism have genetic and biochemical abnormalities in a sleep-related enzyme, according to research presented Friday at a satellite conference of the Society for Neuroscience annual meeting in Washington, D.C.

In high-functioning adults with autism, the signal-to-noise ratio in the outer regions of the brain is significantly lower than in healthy controls, according to unpublished research presented Friday in Washington, D.C.

Individuals who have autism show distinct patterns of gene expression in neurological pathways compared with their unaffected siblings, according to unpublished work presented Saturday at the 2011 Society for Neuroscience annual meeting in Washington, D.C.