Researchers have created detailed three-dimensional reconstructions of the numerous complex branches of dendrites, the signal-receiving ends of neurons.

Mice lacking a gene that regulates an important signaling pathway in the central nervous system have severe autism-like social deficits, including little interest in nurturing their offspring and problems with learning and memory.

Charcot-Marie-Tooth disease and autism are both associated with alterations in the number of copies of certain genetic regions, mutations in multiple candidate genes and with both inherited and spontaneous mutations, notes human geneticist James Lupski.

A protein that regulates the development of serotonin-producing neurons in the brain is associated with autism.

Methylation marks were thought to be permanent features of the genome, but a new study challenges that idea.

The more we find out about human genetics, the more valuable model organisms are likely to become, say autism researchers.  

Boys who have autism-like social deficits at 2 years of age retain about the same level of social impairment when they reach age 20.

Two new studies suggest that people with autism don’t all have trouble detecting the motion of people and animals. What they do struggle with is picking up social information from bodies in motion.

Neurons in a mouse model of fragile X syndrome make more connections during a critical period in development compared with controls, but are slower to respond to signals.

Half of behavioral neuroscience studies published in top-tier journals make a crucial statistical error.