Toddlers with autism are less likely to use gestures to communicate than are typically developing children and those with developmental delay or specific language impairment.

A complicated interplay between a certain genetic variant and autism, schizophrenia and rheumatoid arthritis adds one more intriguing correlation to the growing list of autism risk factors.

A new technique allows researchers to make stable modifications to neurons by incorporating modified amino acids into the proteins of stem cells. The method permits researchers to investigate neuronal function in live, intact cells.

Genetic variants in two autism-associated genes have been linked to repetitive behavior and hyperactivity, two common symptoms of the disorder, according to two studies published in the past few months.

Giving GLYX-13, a drug that targets an autism-associated brain pathway, to rats bred to be less social increases how much they communicate while playing.

The first study to sequence more than 100 genes on the X chromosome in people with autism or schizophrenia has turned up some promising leads.

At a workshop where some of the top scientists in autism research assembled to discuss biomarkers, a 7-year-old girl with the disorder was the real expert.

The protein missing in people with fragile X syndrome regulates the activity of more than 800 other proteins, including some key players in autism, according to a study published 22 July in Cell. Many of these autism-associated proteins cluster on either side of the synapse, the junction between neurons.

Small fragments of RNA, called microRNAs, can fine-tune the levels of proteins at the junctions between neurons in response to cell signals, according to a study published 10 June in Molecular Cell.

Neurofeedback studies showing improvement in symptoms in people with autism are deeply flawed, according to a new review.