Drug counteracts effects of doubled autism gene
An experimental drug tamps down the expression of a gene duplicated in an autism-related condition and restores typical behavior in mice.
An experimental drug tamps down the expression of a gene duplicated in an autism-related condition and restores typical behavior in mice.
Turning down the expression of a gene linked to autism leaves zebra finches singing disorganized songs.
Exposure to infection in utero may speed up the expression of many genes linked to autism — and hasten changes in brain anatomy.
Researchers have identified mutations within regulatory regions that are likely to contribute to autism, using a speedy system of DNA ‘barcodes.’
A new method uses mutations in DNA to maintain a detailed record of series of cellular events.
New findings hint at why drugs that work in mouse models of fragile X syndrome have not been effective in people.
Molecular biologist Jonathan Mill is probing gene expression’s role in autism in uncharted territory — the fetal human brain.
SETD5, a leading candidate for autism risk, may control thousands of genes by modifying a protein involved in packaging DNA.
New findings call into question a fundamental assumption about how genes are regulated.
An online resource reveals all major cell types in the developing human brain during the period in which autism is thought to arise.