Treatments that counteract the effects of an SCN2A mutation in mice increase the animals’ sociability in adulthood, according to a new unpublished study.

A technique that expands tissue before spotting and sequencing RNA — the molecular messenger for DNA — enables researchers to see where particular genes are expressed in cells.

Mutations in CUL3, a gene strongly linked to autism, may cause differences in brain structure by disrupting cytoskeleton proteins, according to a new study.

A new method automatically reconstructs the 3D branching shapes of neurons from images.

The timing of treatment is crucial for conditions related to autism — and more so for some conditions than for others.

Mice that lack a segment of chromosome 22 — a mutation associated with autism — have unusually sparse connections between brain regions.

A new technique leads to neuron cultures of consistent quality, enabling scientists to study how autism mutations alter neurons.

Two imaging techniques together reveal architectural features of the brains of preterm infants.

A top autism gene called SCN2A plays a role at neuronal connections into adulthood, offering hope for treating mutations after infancy.

Mice missing TAOK2, a gene in a segment of chromosome 16 linked to autism, have big brains, immature neuronal junctions and asocial behavior.