Siblings of children with autism who show no signs of the disorder may be compensating with increased activity in two brain regions that detect social cues, according to results presented yesterday at the Society for Neuroscience annual meeting in San Diego.

A combination approach may hold promise for treating repetitive motor behaviors, such as those seen in people with autism, according to research presented yesterday at the Society for Neuroscience annual meeting in San Diego.

A Pittsburgh group has created a comprehensive database of brain scans and other medical and demographic data from nearly 800 individuals whose brains have been injured by strokes. The researchers showcased the collection, called the Western Pennsylvania Patient Registry, at a poster session yesterday at the Society for Neuroscience annual meeting in San Diego.

The Society for Neuroscience annual meeting is the premier event for any scientist interested in the brain. And that’s no less true for those interested in autism research.

A new mouse model for Rett syndrome shows that disrupting a single brain messenger can produce nearly all of the characteristic features of the syndrome, researchers report today in Nature.

Oxytocin may activate the mirror neuron system — a group of neurons that is active when people empathize with others — according to a paper published in the November Psychoneuroendocrinology.

Chromosome 15 may harbor one or more risk genes for autism, according to a new study of multigenerational inheritance in Utah. Researchers have gathered data on family pedigrees stretching back as far as nine generations, with up to five family members affected by the disorder.

Infection early in pregnancy is more harmful to the fetus than at later stages, triggering brain and behavioral changes in the offspring similar to those seen in people with schizophrenia, according to two mouse studies published in October. A third study suggests that exercise can mitigate some of these effects.

An antipsychotic drug often prescribed to treat irritability in children with autism may be more helpful — and cause fewer side effects — depending on an individual’s genetic make-up.

Loss of activity of FOXP1, a member of a family of genes that regulate gene expression, leads to general behavioral defects, including delays in language, according to a study published in November in The American Journal of Human Genetics.