The protein missing in fragile X syndrome, FMRP, facilitates the production of hundreds of unusually large proteins, some of which are linked to autism.

A new method enables scientists to monitor the activity of key molecules involved in the growth and remodeling of connections in the brain.

Seizures early in life may render brain circuits incapable of supporting learning during critical developmental periods.

Many people with fragile X syndrome show average rates of protein production, challenging a long-held assumption about the condition.

A researcher proposes splitting autism into subtypes, mitochondria make neurotransmitters, and highly successful grantees may face a funding cap.

Genes linked to autism are critical to the development of star-shaped brain cells called astrocytes, suggesting a key role for the cells in the condition.

Some genes associated with autism may influence the thickness of the cerebral cortex, the brain’s outer layer.

Microglia come into frequent contact with synapses, the connections between neurons, but they appear to nibble on them rather than engulf and digest them.

Women describe relief at finally learning they have autism, a man with epilepsy narrates during stimulation of his brain, and the brain’s immune cells are caught on film nibbling at neuronal connections.

Deleting one copy of a gene called MVP impairs the brain's ability to adapt to changes in the environment.