The largest genetic analysis of postmortem brain tissue to date has yielded maps of when and where genes related to autism are turned on and off throughout life.

Neurons grown from human stem cells and grafted onto the brains of live mice mature and form connections like those in the fetal human brain.

The autism gene TBR1 controls the expression of several other candidate genes that govern the architecture of the brain’s outer section.

A protective molecular tag on neurons can prevent microglia, the brain’s immune cells, from trimming away their connections with other neurons.

Mice lacking one copy of a leading autism gene have hyperexcitable brains and problems with learning and memory.

Chronic exposure to inflammation in the womb alters autism gene expression and disrupts social behavior in male mice, but not females.

A top autism gene called SCN2A plays a role at neuronal connections into adulthood, offering hope for treating mutations after infancy.

Targeting a fluorescent protein to the cell bodies of neurons enables researchers to clearly see which neurons fire when.

Researchers have charted billions of synapses in the mouse brain and sorted them by type.

The protein missing in fragile X syndrome, FMRP, facilitates the production of hundreds of unusually large proteins, some of which are linked to autism.