Watch the complete replay of André Fenton’s webinar exploring patterns of neural activity that underlie cognition.

Boosting levels of the fragile X protein FMRP in astrocytes reverses features of fragile X syndrome in mice.

Children with autism who have low vasopressin levels struggle to understand the thoughts of others.

Autism is not a developmental disorder, but rather the brain’s adaptive response to early genetic or environmental disturbances, says Mark Johnson.

Glowing particles called ‘Sticky-flares’ can light up RNAs, the molecular blueprints for protein production, in living cells. Using these flares, researchers can plot the paths of specific RNAs through a cell.

Researchers have discovered an enzyme that lowers brain levels of FMRP, the protein missing in people with fragile X syndrome. Blocking the enzyme may ease fragile X symptoms in people with the disorder who have low levels of FMRP and mild symptoms.

Nobel Laureate Tim Hunt resigns after ranting about women in labs, and American scientists blow $28 billion a year on research that can’t be replicated.

The social deficits and repetitive behaviors seen in boys with fragile X syndrome seem to stem from their cognitive difficulties, rather than arising independently as they do in autism.

An autism-linked gene controls the number of neurons in the developing brain. Freda Miller and David Kaplan say the finding points to a new role for the gene in the early embryo.

Researchers are using social media and an online ‘brain-training’ program to study people with rare chromosomal abnormalities linked to autism.