Children with congenital heart defects have increased odds of autism
Children who are born with mild heart problems are more likely to be autistic than their typical peers are.
Autism’s core symptoms accompany a constellation of subtle signs that scientists are just beginning to unmask.
Children who are born with mild heart problems are more likely to be autistic than their typical peers are.
An experimental drug tamps down the expression of a gene duplicated in an autism-related condition and restores typical behavior in mice.
Researchers have used the gene-editing tool CRISPR to disable the MECP2 gene in marmoset embryos. The six such marmosets created seem to recapitulate aspects of Rett syndrome.
Rats missing UBE3A, the gene mutated in people with Angelman syndrome, squeak frequently but tend not to be responsive to the play and squeaks of other rats.
Researchers have created an animated monkey avatar that makes realistic facial expressions — and that may yield insight into how autistic people interpret facial expressions.
A tiny chunk of the brain’s emotion enter, the amygdala, is enlarged in some autistic children; the larger this piece, the more anxious and depressed the child is likely to be.
Delivery by cesarean section leads to subtle brain and behavioral alterations in mice, particularly those delivered prematurely.
Autism and epileptic seizures often go hand in hand. What explains the overlap, and what does it reveal about autism’s origins?
Not much is known about the connection between autism and Ehlers-Danlos syndrome, a condition that affects collagen. But preliminary work provides tantalizing clues.
Studying Smith-Magenis and Potocki-Lupski syndromes — two single-gene conditions in which people have trouble reading social cues — may boost our understanding of autism.