Researchers unveiled a reversible new technique for labeling active neurons in freely moving animals.

The absence of several interacting genes may underlie the developmental problems seen in people missing a segment of chromosome 16.

A protein proves crucial to spatial memory in mice, genes in the 16p11.2 chromosomal region interact in fruit flies’ eye development, and having more autism features tracks with decreased responsiveness to direct gaze.

A new technique illustrates how mutations associated with autism affect gene expression at different stages of development in the fly brain.

Watch the replay of Claudia Bagni’s webinar, in which she described molecular pathways that are impaired in fragile X syndrome, autism and schizophrenia. She also discussed mouse and fly models, as well as human cells.

These short reports from our reporter, Nicholette Zeliadt, give you the inside scoop on developments at the 2015 Dup15q Alliance Scientific Meeting.

Blocking an enzyme involved in learning and memory corrects brain abnormalities and improves memory in fly and mouse models of fragile X syndrome.

A new microscopy technique creates colorful three-dimensional images of brain activity in awake mice.

Experimental drugs can reverse the effects of some autism-linked mutations in the dopamine transporter, a protein that shuttles the chemical messenger dopamine into neurons. Researchers presented the unpublished findings in five posters yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.

Triggering neuron activity with flashes of light allows scientists to tie behaviors in fruit fly larvae to specific neuron groups, reports a study published 25 April in Science. The study catalogs the associations between neurons and the movements they control.