The latest developments in autism research.
People with the autism-linked syndrome lack a protein implicated in several cancers, but it’s unclear whether — or how — they are protected from malignancies.
After a brain transplant of reprogrammed human cells, the animals can for the first time recapitulate some neuronal changes seen in people with fragile X syndrome.
The talk of the Twittersphere turned around two influential neuroscience papers, plus a virtual trip to the World Congress of Psychiatric Genetics.
The technique involves editing the cellular instructions to make MECP2 protein and partially restores its levels in the brainstem.
Tickling our brains on Twitter this week were threads about helping neuroscientists learn the programming language Python, a study about autistic schoolchildren, and the possible root of modern humans’ brain power.
A well-studied brain response to sound appears earlier than usual in young children with autism.
Null and replicated results in this month’s newsletter tackle aging, a purported pathway for oxytocin’s effects on autistic people, and a possible autism biomarker.
The prize recognizes Geschwind’s contributions to our understanding of autism genetics.
A 341-repeat mutation from a person with fragile X does not lead to the syndrome’s traits or function the same way in mice, highlighting a need for different animal models.
The brains of mice carrying different mutations in the autism-linked gene TBR1 display different molecular changes yet similar structural changes, resembling those previously found in autistic people with TBR1 mutations.
Mice with a mutated copy of SHANK3 fail to establish normal sleep patterns during development.
