The results highlight the importance of subgrouping study participants based on their underlying genetics, the researchers say.

Non-neuronal brain cells called astrocytes secrete proteins that seem to hamper the growth of neurons in people with autism-related syndromes. These proteins could be new drug targets, Allen says.

The machine-learning approach could help identify how brain structure differs between autistic and non-autistic infants, the researchers say.

A well-studied brain response to sound appears earlier than usual in young children with autism.

Null and replicated results in this month’s newsletter tackle aging, a purported pathway for oxytocin’s effects on autistic people, and a possible autism biomarker.

The brains of mice carrying different mutations in the autism-linked gene TBR1 display different molecular changes yet similar structural changes, resembling those previously found in autistic people with TBR1 mutations.

Sleep disruption early in life has long-lasting consequences for mice missing a copy of the autism-linked gene SHANK3.

Many brain regions develop differently between people with 22q11.2 duplications and deletions, and those trajectories also vary with a person’s diagnosis.

What these genes do and how they affect autism depends on when in development they’re studied, despite what classic ‘gene ontology’ analyses say.

A delayed brain response to viewing faces may predict lags in social-skill development in autistic people.