Autism and epileptic seizures often go hand in hand. What explains the overlap, and what does it reveal about autism’s origins?
Autism’s core symptoms accompany a constellation of subtle signs that scientists are just beginning to unmask.
Studying Smith-Magenis and Potocki-Lupski syndromes — two single-gene conditions in which people have trouble reading social cues — may boost our understanding of autism.
Not much is known about the connection between autism and Ehlers-Danlos syndrome, a condition that affects collagen. But preliminary work provides tantalizing clues.
A single seizure early in life leads to enduring behavioral problems, including diminished sociability, in mice.
Mouse models of two genetic conditions related to autism show abnormalities in their movement patterns.
Injecting cells called interneurons into the brains of a mouse model of autism restores typical social behavior. But the reason for this effect is a puzzle.
The social brain has a sweet spot that activates when people look each other in the eyes but not when they look at eyes in a video.
A molecule made by mitochondria, the energy factories of cells, might help doctors forecast the impact of mutations in a top autism gene.
Brain activity patterns in the first year of life may predict autism in infants at high risk for the condition.