From funding decisions to scientific fraud, a wide range of societal factors shape autism research.
Screening for metabolic disorders in children with autism is not cost-effective, according to a study published 7 July in PLoS One. The researchers argue instead for careful individual clinical analysis.
Some of the same genetic risk factors may underlie both autism and attention deficit hyperactivity disorder, according to a study published 10 August in Science Translational Medicine.
New studies of restricted and repetitive behaviors link autism to other disorders and may lead to more effective treatments.
Including more females in autism research studies will aid the search for genetic and environmental susceptibility factors for the disorder, says genetic psychiatrist Lauren Weiss.
Children who have more than one older sibling with autism have a one in three chance of developing the disorder themselves, according to a study published today in Pediatrics. The risk is higher for boys: Even if they have only one older sibling with autism, they are almost three times more likely than girls with the same family history to develop the disorder.
Autism studies tend to focus on one part of the spectrum, often excluding those who also have other conditions such as anxiety, obsessive-compulsive disorder or intellectual disability. The result, some experts say, is piecemeal findings that don’t fit together to generate a whole picture.
A new technique allows researchers to make stable modifications to neurons by incorporating modified amino acids into the proteins of stem cells. The method permits researchers to investigate neuronal function in live, intact cells.
The first study to sequence more than 100 genes on the X chromosome in people with autism or schizophrenia has turned up some promising leads.
With a few screen projectors, a pair of three-dimensional glasses and an 8-foot cubicle, researchers have transported adolescents with autism into shaky virtual worlds and discovered that they have surprisingly stable posture.
The protein missing in people with fragile X syndrome regulates the activity of more than 800 other proteins, including some key players in autism, according to a study published 22 July in Cell. Many of these autism-associated proteins cluster on either side of the synapse, the junction between neurons.
