Despite the hype around a new study, it may be too soon to conclude that taking prenatal vitamins during pregnancy lowers the child’s chances of autism.

Understanding the interactions between the placenta and the uterine lining could explain how maternal immune activation leads to autism.

A cell atlas and a cell-culture method promise to advance scientists’ understanding of the placenta and its effects on a fetus.

Consuming Lactobacillus reuteri, a gut microbe found in yogurt and breast milk, may enhance social interactions in three mouse models of autism.

Autism is predominantly genetic in origin, but a growing list of prenatal exposures for mother and baby may sway the odds.

Triggering an immune defense in newborn male mice missing a copy of TSC2, a gene linked to autism, impairs the mice’s social memory.

Chronic exposure to inflammation in the womb alters autism gene expression and disrupts social behavior in male mice, but not females.

Autism results from an interplay between genetics and the environment, but it has been tough to nail down the environmental factors involved.

Rats exposed prenatally to a cocktail of ‘autoimmune’ molecules have altered levels of two types of compounds needed for brain development.

Fever in pregnant rats around the time of delivery disrupts their pups’ communication and social behavior.