Two studies published in the past two months provide new clues to when and how the cerebellum contributes to autism.
David and Bernardo Sabatini, brothers born just a year and a half year apart, invent their way to answering big questions about autism.
Watch the complete replay of Shafali Jeste discussing her work investigating brain structure and function in infants at high risk for autism.
A checklist for fragile X syndrome could help identify people with the condition in low-resource settings, France unveils a plan for early diagnosis and education of children with autism, and virally inserted ‘bar codes’ help track individual neurons.
Mice with the mutations linked to tuberous sclerosis make too little protein in their brains — a finding that contradicts a leading theory about the condition.
New evidence from both people and mice points to a part of the cerebellum that helps process social information as being critical in autism.
A drug called rapamycin prevents seizures in a mouse model of the autism-related condition tuberous sclerosis complex.
Some genes linked to autism regulate the production of proteins at neuronal junctions, suggesting that disrupted protein synthesis contributes to the condition.
Scientists have discovered more than 100 new proteins at junctions in the brain that dampen neuronal activity.