Reactions from SfN 2014
Tune in for daily updates and reactions from attendees at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
Tune in for daily updates and reactions from attendees at the 2014 Society for Neuroscience annual meeting in Washington, D.C.
The brains of children with autism do not prune unneeded connections between neurons as they age, leaving them with too many of these neuronal links, according to a study published 21 August in Neuron.
Watch the complete replay of Robert Malenka’s webinar on the molecular underpinnings of social reward.
The protein missing in fragile X syndrome is necessary for mice to respond to the stimulant cocaine, according to a study published 7 May in Neuron.
Autism, schizophrenia and intellectual disability share underlying deficits in pathways that regulate how the brain encodes new experiences, says Jason Shepherd.
Proteins that help guide neurons to their correct destination in the brain may be involved in autism, says Christopher Cowan.
Long pieces of RNA that do not code for protein have diverse and important roles in the cell and may contribute to autism risk, say Nikolaos Mellios and Mriganka Sur.
Spontaneous and rare mutations, particularly in genes related to networks that regulate neuronal connections, contribute a small but significant proportion of the risk for schizophrenia, report two large studies published online 22 January in Nature.
Watch the complete replay of Michael Merzenich discussing how the brain’s ability to change with experience may lead to new treatments for autism.
Emerging evidence indicates that microglia, the brain’s immune cells, are altered in some individuals with autism, raising questions about their role in brain development, says Beth Stevens.