Antidepressant eases some autism features in mice
A drug that keeps neurons bathed in the chemical messenger serotonin prevents social abnormalities in a mouse model of dup15q syndrome.
A drug that keeps neurons bathed in the chemical messenger serotonin prevents social abnormalities in a mouse model of dup15q syndrome.
A drug that alters the balance of two key chemical messengers in the brain may help treat autism, suggests a proof-of-principle study.
A drug used to treat excessive swelling seems to ease autism features in some children on the spectrum.
Some mutations in a gene called SCN2A make neurons less excitable and are linked to autism; others have the opposite effect and may cause seizures during infancy.
Scientists have discovered more than 100 new proteins at junctions in the brain that dampen neuronal activity.
The brains of people with autism show a distinct molecular signature that reflects alterations in how genes are pieced together and expressed.
Lowering the levels of OTX2, a protein found in the fluid that bathes the brain, prevents many abnormal behaviors in mouse models of Rett syndrome.
Injecting stem cells into young rats makes them more social and flexible when learning tasks than before the treatment.
Cells derived from the skin of boys and men with autism share a host of unusual characteristics.
Manipulating MeCP2, the gene mutated in Rett syndrome, has revealed two neuron types as crucial contributors to the condition.