Mice missing a gene called PTCHD1 in a deep-seated brain structure have autism-like symptoms that ease with treatment.
A new tool allows researchers to simultaneously study the physical, genetic and electrical properties of individual neurons.
A new tool marries an unusually bright fluorescent protein to a light-sensitive pigment to illuminate individual neurons as they fire.
There are hints that transcranial magnetic stimulation, which uses electricity to change how brain cells function, might improve the symptoms of autism. But hopes are running way ahead of the facts.
By analyzing stem cells derived from baby teeth, researchers have tracked a child’s autism to mutations in a gene called TRPC6. The molecular saga highlights a painless way to probe the role some genes play in autism.
Long pieces of RNA that do not code for protein have diverse and important roles in the cell and may contribute to autism risk, say Nikolaos Mellios and Mriganka Sur.
Loss of one copy of the autism-linked gene PTEN in mouse neurons boosts production of a certain ion channel, and decreases the neurons’ firing rate, according to a study published 21 October in Proceedings of the National Academy of Sciences.