Cannabidiol (CBD) blocks the action of a molecule that drives an overexcitability feedback loop in a rodent model of epilepsy.

Cells from people with fragile X syndrome overproduce — but don’t accumulate — proteins. New work suggests that excessive protein breakdown may account for this discrepancy, and explain some of the syndrome’s traits.

Brain scans of the hippocampus reveal autistic people who are at increased risk of cognitive problems as they get older.

A range of presentations at Neuroscience 2022 tie atypical social behavior to trouble discriminating between odors in the animals.

An overabundance of ribosomes drives an imbalance of proteins produced from long and short genetic transcripts in a mouse model of fragile X syndrome.

The animals recall fear more readily if a set of inhibitory neurons in the hippocampus is switched on.

The protein, FMRP, shapes cell signaling near synapses but switches to regulate genes in the cell body, according to new research.

Deleting a copy of the gene TBX1 depletes the fatty myelin insulation that surrounds neurons and reduces cognitive speed in mice.

A potential new gene therapy delivered via a single injection mimics ratios of proteins normally found in cells.

Activating certain receptors in the amygdala — a treatment that runs counter to a leading theory of what causes the condition — can reverse some traits in rats.