Neurons grown from human stem cells and grafted onto the brains of live mice mature and form connections like those in the fetal human brain.
Children who have repetitive behaviors, a core autism trait, may show particular patterns of brain activity as early as 1 year of age.
The autism gene TBR1 controls the expression of several other candidate genes that govern the architecture of the brain’s outer section.
Administering a cholesterol drug alongside an antibiotic eases atypical behavior and restores the signaling balance in the brains of people with fragile X syndrome.
A tiny fraction of the connections between brain regions can identify an individual.
Brain activity governing language can distinguish children with autism into distinct subgroups.
Analyzing large numbers of autism mice, researchers have found that the mice cluster into subtypes based on brain structure and functional connectivity.
Mapping the effects of autism mutations on mouse brain circuits may reveal subtypes of the condition in people.
Looking at the brain as a whole suggests that nudging flawed sets of neurons to collaborate better might alleviate autism traits.
Injecting a virus toting snippets of RNA into the rodent brain enables researchers to express genes in specific neuron types.