The new technique can record hundreds of thousands of neurons firing nearly simultaneously across big swaths of brain tissue in living mice.

Scientists should heed these differences when considering resting-state brain activity as a biomarker for autism, the researchers say.

The first animal model of MYT1L syndrome suggests that fast-maturing neurons lead to the unusually small brains, social deficits and other traits seen in people with the condition.

The X chromosome holds stronger-than-expected genetic sway over the structure of several brain regions. The genes that may underlie this oversized influence have ties to autism.

Four subtypes lend new support to the idea that there isn’t a single ‘hallmark’ type of brain connectivity in people with autism.

Reports of flaws in imaging research are signs of a maturing field, experts say, not a devastating blow to its results.

Atypical patterns of neuronal activity and gene expression in the striatum may characterize autism in girls, according to a new study.