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Mutations in autism gene may be treatable beyond infancy
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A top autism gene called SCN2A plays a role at neuronal connections into adulthood, offering hope for treating mutations after infancy.
A top autism gene called SCN2A plays a role at neuronal connections into adulthood, offering hope for treating mutations after infancy.
Autism and intelligence share genetic variants, researchers grow Neanderthal mini-brains and see overlap with autism, and maternal diabetes is an autism risk factor.
Four new imaging techniques published over the past few months enable researchers to easily visualize cells and other features in tissue slices and living brains.
Spheres of brain cells derived from people and implanted into mouse brains recruit blood vessels and integrate with mouse neurons.
A new map reveals the routes of individual neurons in visual regions of the mouse brain.
The same techniques that generate images of smoke, clouds and fantastic beasts in movies can render neurons and brain structures in fine-grained detail.
A collection of brain scans from monkeys aged 2 weeks to 12 months reveals how their brain structures and nerve tracts develop over time.
A clue to the origins of autism may exist among the molecules that transport RNA in the developing brain.
A mutant virus that infiltrates neurons and delivers genes to the nucleus can illuminate brain connections.
A pair of existing drugs normalizes the appearance and activity of neurons derived from the skin of individuals with Rett syndrome.