In a video interview at the 2012 Society for Neuroscience annual meeting, Edward Boyden discusses new tools his lab is developing to refine optogenetics techniques.

Mice missing the autism-linked gene CNTNAP2 have broad defects in their oxytocin system, according to research presented Tuesday at the Society for Neuroscience annual meeting in New Orleans. Treatment with oxytocin alleviates social deficits in these mice.

The brain’s response to biological motion can distinguish typically developing children from those who have a sibling with autism but do not have the disorder themselves, according to research presented at the 2012 Society for Neuroscience annual meeting in New Orleans.

A cholesterol-lowering drug called lovastatin prevents seizures in mice that model fragile X syndrome, according to a poster presented Sunday at the 2012 Society for Neuroscience annual meeting in New Orleans.

A new microscopy technique allows researchers to take high-resolution three-dimensional images of intact mouse brains.

Rats lacking FMR1, the gene mutated in people with fragile X syndrome, do not learn to discriminate between human speech sounds like control rats do, according to research presented Monday evening at the 2012 Society for Neuroscience annual meeting in New Orleans.

Sensory illusions are much more than just parlor tricks or diversions, says Carissa Cascio. In a video interview with SFARI.org at the Society for Neuroscience annual meeting, she explains why sensory illusions work, and what they might be able to teach us about autism.

The first wave of data from the Human Connectome Project, a five-year $30 million effort to map the structure of the human brain, is now freely available, researchers announced at the 2012 Society for Neuroscience annual meeting in New Orleans.

Children with autism are less susceptible to an illusion involving sight and touch, according to unpublished research presented Monday at the Society for Neuroscience annual meeting in New Orleans.

Individuals with an extra copy of MeCP2, the gene mutated in Rett syndrome, have severe developmental delay accompanied by seizures, respiratory infections, poor motor skills and features of autism, according to two new case studies.