Researchers are studying how a combination of genetic ‘hits’ may contribute to autism’s diversity.
Autism may be just as common among children missing a segment of chromosome 16 as it is in those with an extra copy.
A cellular pathway that helps neurons grow and move during fetal development may drive the changes in head size in some autistic people.
Clusters of brain cells — so-called ‘mini-brains’ grown in the lab — may help researchers understand how large stretches of duplicated or deleted DNA affect the brain.
Mapping the effects of autism mutations on mouse brain circuits may reveal subtypes of the condition in people.
The absence of several interacting genes may underlie the developmental problems seen in people missing a segment of chromosome 16.
Boosting the levels of the chemical messenger serotonin makes mice that model autism more social — and brings new hope to a shelved theory in autism.
New results implicate an enzyme called ERK in the consequences of missing a segment of chromosome 16.