In this edition, researchers sink a purported link between cerebellar volume and autism and buoy a theory about measuring social behaviors.

ADNP and SHANK3 proteins may bind together and alter a neuron’s internal scaffold, hinting at a mechanism that, when disrupted, may underlie several forms of autism.

By as early as age 2, autistic children appear to have a smaller salience network and a larger default mode network, among other differences, than children without the condition.

Neurons with a faulty copy of SETD1A, a gene tied to autism and schizophrenia, show structural abnormalities and altered connectivity patterns.

On Twitter this week, many researchers toasted the geneticists who won the 2022 Kavli Prize in neuroscience, and others offered helpful reminders for the field.

Different combinations of common, rare, inherited and spontaneous mutations may explain why traits vary so widely among autistic people.

Strategies to replace or compensate for mutated copies of the TCF4 gene could lead to treatments for this profound form of autism, a new study suggests.

The chair and co-chair of the Student and Trainee Committee for the International Society for Autism Research share their advice for early-career researchers.

Drugs such as LSD act primarily on the serotonin system, which is implicated in autism — and some autistic people who experiment with psychoactive compounds report enhanced social connections, among other benefits. But researchers have more questions than answers.

This month’s newsletter also highlights deflated autism prevalence estimates from Shanghai, China.