Deleting a neuronal protein associated with autism causes oxidative stress — characterized by an excess of free radicals — which has been linked to diseases such as diabetes and Alzheimer’s, according to new research in worms. The results were presented yesterday at a poster session at the Society for Neuroscience meeting in Chicago.
People born without the large bundle of nerve fibers that bridges the brain’s hemispheres have trouble identifying fearful faces, and don’t look preferentially at others’ eyes to perform this task, according to research presented Sunday at the Society for Neuroscience meeting in Chicago.
On Saturday, a top government official resigned from the Interagency Autism Coordination Committee, the body of scientists and advocates that’s responsible for guiding all autism research funded by the U.S. National Institutes of Health. Institute director Francis Collins responds.
Mice carrying an autism-associated mutation show impaired social interactions and dramatic changes in brain size when their immune systems are activated, according to research presented yesterday at a poster session at the Society for Neuroscience meeting in Chicago.
After a Monday afternoon poster session at the Society for Neuroscience meeting in Chicago, Noboru Hiroi talked about the challenges of following up human genetic findings in the mouse.
A gene tied to autism and language impairment is crucial for the early development and migration of inhibitory interneurons, according to research presented Monday at the Society for Neuroscience meeting in Chicago.
New pharmacological and behavioral interventions can reverse characteristics of autism in a mouse model of the disorder, according to unpublished results presented in poster sessions today at the Society for Neuroscience meeting.
National Institutes of Health director Francis Collins talks about the agency’s upcoming plans for “beefing up” autism research, including more than $100 million each year in grants for the field.
Adolescents who carry certain common variants in a gene associated with autism — whether they have the disorder or not — show abnormal connectivity between brain regions, according to unpublished data presented at the Society for Neuroscience meeting in Chicago.
Two variants in a gene needed for the cell’s energy balance may protect the carriers from autism, according to an unpublished study presented today at the Society for Neuroscience meeting in Chicago.