Chromatin remodeling tied to altered splicing in autism model
Exposing neurons to valproic acid, a well-known environmental risk factor for autism, disrupts their ability to generate different proteins from the same gene.
Exposing neurons to valproic acid, a well-known environmental risk factor for autism, disrupts their ability to generate different proteins from the same gene.
High school interns don’t just gain new skills — they also form a pipeline of future scientists.
Connections between 13 autism-linked proteins and their binding partners in excitatory neurons implicate a new molecular pathway.
Discussion among autism researchers on Twitter this week swirled around how well pediatricians assess the condition, how it affects cognitive aging and how it alters cardiometabolic disease risk.
The findings put genetic background forward to help explain autism’s heterogeneity.
Elsevier’s retractions focus on peer review and conflicts of interest.
The gene, linked to a little-known condition called Weiss-Kruszka syndrome, prevents embryonic stem cells from deviating from their neuronal destiny.
Altered expression of TSC2 and the mTOR pathway reshape the formation of certain synapses between inhibitory and excitatory neurons in mice.
Conversation took off this week around precision in neuroscience measurements, a new strategy to rescue Rett neurons, autism insights from protein interactomes, and mechanisms of touch perception.
CRISPR-edited prairie voles that lack receptors for the so-called “social hormone” still bond with their mate and pups, raising questions about the molecule’s role.