Varying the number of copies of a single autism-linked gene modulates social behavior and aggression in mice, according to unpublished results presented yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.

Scientists reflect on the current state of autism research as the 2014 Society for Neuroscience annual meeting in Washington, D.C. comes to a close.

A rare form of autism linked to a duplication of the 15q11-13 chromosomal region shares a molecular signature with more common forms of the disorder, suggests unpublished research presented yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.

Scientists have created a transgenic monkey modeling Rett syndrome, they announced yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C. This model and others sparked a lively discussion about the relative value of animal models in research.

The expression patterns of microRNAs vary less by brain region and age in people with autism than in controls. Researchers presented the unpublished findings Tuesday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.

Robots that help children with autism become more socially engaged may also increase understanding of sensory processing in the disorder, suggests unpublished research presented today at the 2014 Society for Neuroscience annual meeting in Washington, D.C.

Loss of MeCP2, the Rett syndrome gene, depletes immune cells throughout the bodies of mice, researchers reported yesterday at the 2014 Society for Neuroscience annual meeting in Washington, D.C.

Monkeys living in natural groups show individual variations in social behavior that may help scientists understand autism and identify treatments for the disorder, according to unpublished studies presented at the 2014 Society for Neuroscience annual meeting in Washington, D.C.

Mice born to older males with mutations in PAX6 — a gene involved in brain development — vocalize less than those with younger dads. The unpublished findings, presented today at the 2014 Society for Neuroscience annual meeting in Washington, D.C., suggest how genes and paternal age can work together to trigger symptoms.

The memory and sleep troubles that accompany fragile X syndrome originate in a glitch in insulin signaling, suggests an unpublished study of fruit flies presented today at the 2014 Society for Neuroscience annual meeting in Washington, D.C. The study points to a widely available diabetes treatment for the syndrome.