Fever’s immune effect on brain may ease autism traits
An immune molecule produced during a fever improves sociability in three mouse models of autism.
An immune molecule produced during a fever improves sociability in three mouse models of autism.
The many genes implicated in Williams syndrome, a condition related to autism, may all work together to exert their effects on behavior.
A shortage of CUL3, a leading autism gene, may impair social behavior in mice by ramping up protein production in neurons.
Some cases of anxiety, which often accompanies autism, may stem from a faulty immune system, a new study suggests — but some experts are skeptical.
The drug popularly known as ecstasy may boost sociability through brain circuits distinct from that underlying its ‘high.’
Extra copies of a gene called BOLA2 predispose people to autism and may protect against iron deficiency.
People who have mutations in a gene called PHF21A tend to have a constellation of traits and conditions, including autism.
People with attention deficit hyperactivity disorder may carry certain rare, harmful mutations in many of the same genes as people with autism.
Blocking one form of an enzyme implicated in autism eases unusual behaviors and seizures in mice missing a top autism gene.
Children born with high blood levels of vitamin D have 25 percent decreased odds of autism compared with those born with low levels.